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Study: Defective EBV Virus is the Cause of Chronic Fatigue Syndrome (CFS); polyDNA Recommends Gene-Eden-VIR and Novirin to Reduce Fatigue Linked to a Viral Infection

>PRWEB.COM Newswire

Rochester, NY (PRWEB) May 10, 2014

Extensive research shows a link between EBV and Chronic Fatigue Syndrome (CFS). However, researchers wanted to go beyond observing a correlation. They wanted to understand the molecular mechanism that leads from an EBV infection to Chronic Fatigue Syndrome. Over many years, scientists proposed many different mechanisms. In all of them, the active EBV played a major role. Now, scientists report some unusual findings. Researchers wanted to know "if people with chronic fatigue syndrome were mounting an effective immune response against EBV. They also looked for direct evidence of an active EBV infection (1)." Surprisingly, they found "no evidence of active EBV infection (1)" in CFS patients.

So how is EBV linked to CFS?

"One theory, developed by Dr. Lerner and a group at The Ohio State University (that includes Drs. Ariza, Glaser, and Williams), proposes that EBV undergoes 'abortive replication' in some people with ME/CFS. In abortive replication, a defective form of EBV produces early proteins, but is unable to produce later ones. The Ohio State group believes continual production of these proteins is causing a chronic inflammatory state in some people with ME/CFS (1)."

Another theory that shares some ideas with abortive replication was presented by Dr. Hanan Polansky, in 2003. (See (2). According to Dr. Polansky, during the EBV latency phase, the virus still produces proteins. The viral DNA which includes sequences called N-boxes that cause most major diseases. The viral N-boxes microcompete with the human DNA and can causes the human DNA to produce abnormal levels of human proteins. This leads to disease.

In both theories, the human disease is caused by the production of a limited set of viral proteins, a set which is different from that of a fully functioning active virus. In other words, both theories do need the virus to be active for a disease process to start. They only need the presence of the virus, and the production of a limited set of proteins. Dr. Polansky's theory uses the common term, "latency", while Dr. Lerner's theory uses the term "abortive replication."

To learn more about Dr. Hanan Polansky's research and the Theory of Microcompetition with Foreign DNA, visit: (2).

Chronic Fatigue Syndrome is a "devastating disease that still puzzles doctors, and only a fraction of sufferers are properly diagnosed. Once derided as 'yuppie flu,' CFS actually cuts across all races and economic groups. But it still battles a heavy stigma, and research funding is scarce. Thirty years after its first discovery, there is no single diagnostic test and no treatment (3)."

The CDC says that "Chronic fatigue syndrome, or CFS, is a devastating and complex disorder. People with CFS have overwhelming fatigue and a host of other symptoms that are not improved by bed rest and that can get worse after physical activity or mental exertion. They often function at a substantially lower level of activity than they were capable of before they became ill." (See the CDC's web page on CFS, last reviewed on May 14, 2012) (4).

"We recommend that if you suffer from chronic fatigue, you should get an EBV blood test. If positive, talk to your doctor about Gene-Eden-VIR and Novirin." - Mike Evans, polyDNA

As far as we know, the only two products currently available that target latent viruses are Gene-Eden-VIR, and Novirin.

Novirin shares the same formula as Gene-Eden-VIR. The difference between the two is that Novirin has higher quality, more expensive ingredients. The Novirin/Gene-Eden-VIR formula was tested in two post-marketing clinical studies published in September 2013 and March 2014, respectively, in the peer reviewed medical journal Pharmacy & Pharmacology (5).

Interested individuals can view the two published studies here, and

Up to 70% of those studied reported a decrease in symptoms associated with CMV infection, and users of the Novirin/Gene-Eden-VIR formula experienced an increase in overall health (5).

Each ingredient of Novirin was chosen through a scientific approach. Scientists scanned thousands of scientific and medical papers published in various medical and scientific journals around the world to identify the highest quality, safest, most effective natural ingredients that target latent viruses.

A second clinical study showed that the Novirin/Gene-Eden-VIR formula decreased physical and mental fatigue. (See Pharmacology & Pharmacy, from March, 2014) (6).

To learn more about Novirin, visit and about Gene-Eden-VIR, visit

All orders of these products are completely confidential, and no information is shared or sold to any third party. Privacy is assured.


(1) EBV I: A Deficient Immune Response, Increased Levels of Epstein-Barr Virus Opens Up EBV Question in Chronic Fatigue Syndrome Again. Published on March 10, 2014.

(2) The Center for the Biology of Chronic Disease (CBCD)

(3) Chronic fatigue syndrome activists launch 'uprising' from their beds. Published on January 3, 2014

(4) CDC - Chronic Fatigue Syndrome (CFS) - General. Last Reviewed on May 14, 2012.

(5) Gene-Eden-VIR Is Antiviral: Results of a Post Marketing Clinical Study. Published in September 2013.

(6) Eden-VIR Decreased Physical and Mental Fatigue in a Post Marketing Clinical Study That Followed FDA Guidelines; Results Support Microcompetition Theory. Published in March 2014.

polyDNA is a biotechnology company that develops dietary supplements using the unique scientific method developed by Dr. Hanan Polansky, which is based on Computer Intuition.

In addition to his unique scientific method, Dr. Polansky published the highly acclaimed scientific discovery, called Microcompetition with Foreign DNA. The discovery explains how foreign DNA fragments, and specifically, DNA of latent viruses, cause most major diseases.

polyDNA developed Novirin, an antiviral natural remedy that helps the immune system kill latent viruses.

Read the full story at

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